No matter if you live in an apartment or a house or simply need to ensure that your house is free of EMFs There are a variety of ways you can reduce exposure. One of the easiest is to limit your electronic device use. It is also possible to use EMF block paint to prevent EMF radiation from reaching your home. Another way to shield your house against EMF radiation would be to install a RF shielding canopy. It is a type of net that contains EMF shielding. It's used to prevent EMFs from entering a space. Another alternative is to have your house equipped with an enclosure that is conductive. These devices are called Faraday cages.
Numerous studies have demonstrated how the EMF that is not ionized has antiproliferative properties in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is thought to result from the deregulation the cancer stem cell. This could explain the long-term response seen in patients with advanced HCC. However, the mechanism of AM EMF's impact on patients with cancer is not clear.
Aspects of AM RF EMF on HCC tumor growth in vivo were studied in mice. The tumors were classified into three groups. First, emf blocking clothing that was unaffected to RF EMF. Second group members were exposed RF EMF at frequencies similar to that of humans. In the third, they were subjected RF EMF with HCC-specific modulation frequencies. The effect of HCCMF on the tumours was assessed against the effect of RCF. The results indicated that tumours treated with HCCMF showed significant shrinkage. However, the tumours treated with RCF showed no evidence of shrinkage of the tumor.
The reason for tumour-specific AM RF EMF might be based on the fact that tumor cells require Cav3*2 T-type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation on HCC cells is caused by CACNA1H the protein which mediates tumour-specific Ca2+ influx. The results suggest that CACNA1H may have broader implications for the diagnosis and treatment of a variety of cancers.
The tumours of the controls were never exposed to EMF from radiofrequency, and fed a standard mouse diet. The tumors in the HCCMF group were injected with Huh7 cells when they were between five and seven weeks old. The tumors were then killed when they showed excessive burden.
The tumors of the three groups also displayed distinct growth curves. emf radiation blocker -treated tumors had a significant reduction in tumour size after eight weeks. However, the tumours which were treated by RCF showed no reduction in size. The difference was significant. The tumors treated by RCF had necrosis that is common in tumors that have been exposed to RCF. It is possible that this necrosis is caused by a lack of oxygen in the larger cancers.
In summary, the results suggest an AM-RF EMF is a powerful source of anti-cancer properties in vitro as well as in the vivo. block emf have demonstrated that AM RF EMF produces measurable tumour shrinkage within HCC patients. The possibility is that the AM EMF triggers these effects because of CACNA1H, a protein involved in the tissue-specific Ca2+ influx. In addition, AM RF EMF may exert a sustained impact on the development of HCC tumours in living tissue.